Synaptotagmin 7 switches short-term synaptic plasticity from depression to facilitation by suppressing synaptic transmission
Abstract Short-term synaptic plasticity is a fast and robust modification in neuronal presynaptic output that can enhance release strength to drive facilitation or diminish it to promote depression.The mechanisms that determine whether neurons display short-term facilitation or depression are still unclear.Here we show that the Ca2+-binding protein